Factory For Tylosin Tartrate injection 10%*50ml,100ml Wholesale to Angola

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  • Price & Quotation: FOB Shanghai: Discuss in Person
  • Shipment Port: Shanghai, Guangzhou, Chongqing, Yiwu
  • MOQ(5ml,10ml): 30000 Bottles
  • MOQ(50ml,100ml): 5000 Bottles
  • MOQ(250ml,500ml): 2000 Bottles
  • MOQ: Powder/Bolus: 500 KG
  • Payment Terms: T/T, L/C
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    Factory For Tylosin Tartrate injection 10%*50ml,100ml Wholesale to Angola Detail:

    Composition:

    Each 1 ml contains Tylosin Tartrate 200mg.

    Descreptions: N/A

    Indications:

    Treats shipping fever, pneumonia, foot rot, calf diphtheria, metritis in beef cattle and non-lactating dairy cattle. Treats mycoplasmal swine arthritis, pasteurella pneumonia, erysipelas, acute swine dysentery in swine.

    Pharmacological action:

    Tylosin is thought to have the same mechanism of action as ery�thromycin (binds to 50S ribosome and inhibits protein synthesis) and exhibits a similar spectrum of activity. It is a bacteriostatic antibiotic. For more specific information on or�ganisms that tylosin is usually active against, refer to the erythromycin monograph just prior to this one. Cross resistance with erythromycin occurs.

    Usage and administration:

    Beef and non-lactating dairy cattle: Inject IM 8 mg per pound of body Weight (1 mL per 25 pounds) once daily. Treatment should be continued 24 hours after symptoms of the disease have stopped, not to exceed 5 days. Do no inject more than 10 ml per injection site.

    Swine: Inject IM 4 mg per pound of body weight (1 mL per 50 pounds) twice daily. Treatment should be continued 24 hours after symptoms of the disease have stopped, not to exceed 3 days. Do not inject more than 5 mL per injection site.

    Precaution:

    Do not mix Tylosin Injection with other injectable solution as this may cause precipitation of the active ingredient. Do not administer to horses or other equine species. Injection of tylosin in equines has been fatal.

    Withdrawal time:

    Discontinue use in cattle 21 days before slaughter. Discontinue use in swine 14days before slaughter.

    Storage and expired time:

    Put in a cool dry place away from direct sunlight. Store in a cool, dry place under 22 �C, away from direct sunlight.

    2 years

    Packing:50ml,100ml


    Product detail pictures:

    Factory For
 Tylosin Tartrate injection 10%*50ml,100ml Wholesale to Angola detail pictures

    Factory For
 Tylosin Tartrate injection 10%*50ml,100ml Wholesale to Angola detail pictures

    Factory For
 Tylosin Tartrate injection 10%*50ml,100ml Wholesale to Angola detail pictures

    Factory For
 Tylosin Tartrate injection 10%*50ml,100ml Wholesale to Angola detail pictures


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    Teeth whitening Hull

    Tooth whitening (termed tooth bleaching when utilising bleach), is either the restoration of a natural tooth shade or whitening beyond the natural shade.

    Restoration of the underlying natural tooth shade is possible by simply removing surface stains caused by extrinsic factors, stainers such as tea, coffee, red wine and tobacco. The buildup of calculus and tartar can also influence the staining of teeth. This restoration of the natural tooth shade is achieved by having the teeth cleaned by a dental professional (commonly termed “scaling and polishing”), or at home by various oral hygiene methods. Calculus and tartar are difficult to remove without a professional clean.

    To whiten the natural tooth shade, bleaching is suggested. It is a common procedure in cosmetic dentistry, and a number of different techniques are used by dental professionals. There is a plethora of products marketed for home use to do this also. Techniques include bleaching strips, bleaching pens, bleaching gels and laser tooth whitening. Bleaching methods generally use either hydrogen peroxide or carbamide peroxide which breaks down into hydrogen peroxide. Common side effects associated with bleaching include increased sensitivity of the teeth and irritation of the gums.
    Natural tooth shade

    1: crown, 2: root, 3: enamel, 4: dentine and dentine tubules, 5: pulp chamber, 6: blood vessels and nerve within root canal, 7: periodontal ligament, 8: apex and periapical region, 9: alveolar bone
    The perception of tooth color is the result of a complex interaction of factors such as: lighting conditions, translucency, opacity, light scattering, gloss, the human eye and brain.[1] Teeth are composed of a surface enamel layer, which is whiter and semitransparent, and an underlying dentin layer, which is darker and less transparent. These are calcified, hard tissues comparable to bone. The natural shade of teeth is best considered as such; an off-white, bone-color rather than pure white. Public opinion of what is normal tooth shade tends to be distorted. Portrayals of cosmetically enhanced teeth are common in the media. In one report, the most common tooth shade in the general population ranged from A1 to A3 on the VITA classical A1-D4 shade guide.[2][3]

    Females generally have slightly whiter teeth than males, partly because females’ teeth are smaller, and therefore there is less bulk of dentin, partially visible through the enamel layer. For the same reason, larger teeth such as the molars and the canine (cuspid) teeth tend to be darker. Baby teeth (deciduous teeth) are generally whiter than the adult teeth that follow, again due to differences in the ratio of enamel to dentin. As a person ages the adult teeth often become darker due to changes in the mineral structure of the tooth, as the enamel becomes less porous[citation needed] and phosphate-deficient. The enamel layer may also be gradually thinned or even perforated by the various forms of tooth wear.

    Tooth staining and discoloration
    Main article: Tooth discoloration

    Internal resorption of the left maxillary lateral incisor (right in photograph), giving rise to the appearance termed “Pink tooth of Mummery”
    Teeth may be darkened by a buildup of surface stains (extrinsic staining), which hides the natural tooth color; or the tooth itself may discolor (intrinsic staining).[4]

    Extrinsic discolouration
    Extrinsic stains can become internalised through enamel defects or cracks or as a result of dentine becoming exposed but most extrinsic stains appear to be deposited on or in the dental pellicle.[5] Causes of extrinsic staining include:

    Dental plaque: although usually virtually invisible on the tooth surface, plaque may become stained by chromogenic bacteria such as Actinomyces species.[6]
    Calculus: neglected plaque will eventually calcify, and lead to the formation of a hard deposit on the teeth, especially around the gumline. The color of calculus varies, and may be grey, yellow, black or brown[6]
    Tobacco: tar in smoke from tobacco products (and also smokeless tobacco products) tends to form a yellow-brown-black stain around the necks of the teeth above the gumline[6]
    Betel chewing.[7]
    Certain foods and drinks. food-goods and vegetables rich with carotenoids or xanthonoids.[citation needed] Ingesting colored liquids like sports drinks, cola, coffee, tea, and red wine can discolor teeth.[8]
    Certain topical medications. Chlorhexidine (antiseptic mouthwash) binds to tannins, meaning that prolonged use in persons who consume coffee, tea or red wine is associated with extrinsic staining (i.e. removable staining) of teeth.[9]
    Metallic compounds. Exposure to such metallic compounds may be in the form of medication or other environmental exposure. examples include iron (black stain), iodine (black), copper (green), nickel (green), cadmium (yellow-brown).[4]
    Intrinsic discolouration
    Changes in the thickness of the dental hard tissues would result in

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